11 The reported distribution
The second family is the ensemble-level report: the histogram in the appendix, the SD in the norms table, the curve a percentile is read off. It is the family the flexible prior exists to serve, and on these cases it is where the prior’s consequence concentrates, with a twist that the score results make sharp: the distribution moves most exactly where scores move least.
11.1 Materiality inverts between families
Of the seven bimodal case-cells, one was material on individual scores; on the reported distribution, all seven are, with a median between-prior KS of 0.076. The skewed cells are material nine of nine (median KS 0.106), the normal controls two of five with a median at 0.048 against the 0.05 bar, controls behaving as controls. Under bimodality, then, the prior changes the reported distribution without moving the people in it. A reader who checked a handful of individual scores, found them unchanged, and concluded the prior did not matter would be wrong about the norms table they were about to publish; Figure 11.1 shows the phenomenon on C4 directly, with the summary swap beside it for scale (on this family the two levers are both material almost everywhere, and which displaces the curve farther is the reliability-governed question Chapter 9 settled).
11.2 A family change, not a rescale
The moments of the reported ensembles say what kind of change this is, and they say it only if read together. On the skewed cells the DP arm’s ensemble has a larger standard deviation (ratio 1.064) and a smaller interquartile range (ratio 0.925), and its excess kurtosis moves from -0.17 under the Gaussian to +1.14. More spread overall, less spread in the middle, is not a rescaling; it is one distribution family being replaced by another, a platykurtic shrunken ensemble giving way to a leptokurtic one with a tight core and heavy tails. The bimodal cells show the same signature more weakly in spread (SD ratio 1.031, IQR ratio 0.994) and clearly in shape (kurtosis -0.04 to +0.77). Figure 11.2 places every case-cell on this plane.
Had this book reported only the SD ratio, the finding would read “the DP prior disperses the ensemble by about 6%”; only the IQR ratio, “the DP prior tightens the ensemble by about 8%”. Both sentences are true and neither is the finding, which is why the comparison layer refuses to collapse F2 to any single spread statistic, and why it refused from the start to standardize the outputs before comparing them: a standardizing step sets the SD ratio to one by construction and would have converted this signature into an artifact pointing the wrong way. Figure 11.3 runs the check the refusal implies: after standardizing each ensemble within itself, the two priors’ curves still separate on the non-normal cases and coincide on the control. What remains after location and scale are removed is the part no affine correction can supply.
11.3 What the flexible prior estimated
The reported ensembles are summaries; the object the DP arm actually estimates is the latent density itself, with uncertainty. Figure 11.4 shows it for four focal cases with pointwise 95% bands, and it earns a caution the first edition relegated to a footnote. The display is deliberately non-like-for-like: the DP line is a posterior latent-density estimate, whereas the Gaussian reference is a normal curve fitted to the shrunken Gaussian-PM ensemble. It illustrates a reporting contrast, not a density-estimator contest. The DP curve is a posterior mean over 16,000 draws, and averaging smooths: a population the shape screen classifies as bimodal from its score pattern can present a unimodal posterior-mean density (C4’s shoulder is visible, C10’s valley shallow), so the panel illustrates what the prior learned, not the coordinate of record. On C12 the band is wide and the flexible density hugs the Gaussian’s: at reliability .43 the data cannot tell the prior much either way, a picture worth holding until Chapter 17.
11.4 What this family establishes
The prior’s consequence concentrates in the reported distribution, and under bimodality it appears there while scores hold still: whether the prior “matters” has no dataset-level answer, only a family-level one. The change is a family change (SD up, IQR down, kurtosis through zero) that any single spread statistic misreports in one direction or the other. Percentile tables and norms built from these ensembles differ between priors even where every individual’s score agrees to two decimals. What this family does not establish, on any case, is which reported distribution is closer to truth. Part V reads the simulation’s answer at the cells these tests occupy, under a join that is exploratory because it was assembled after both source volumes’ results; the direction it reports is the simulation’s, never the case’s.



